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These agents are not absorbed and work best for patients with GI symptoms or those whose toxin exposure is coming from food. Sequestrants bind to toxins in the GI tract making them unavailable for reabsorption. Transdermal and liposomal glutathione may also be helpful, especially in combination with sequestrants.
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Nebulized and intranasal glutathione is beneficial for those exposed to inhaled toxin.
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Exposure to VRA can cause immunological problems, vomiting, skin dermatitis, and hemorrhagic lesions.
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VRA causes damage to human cells by inhibiting protein and DNA synthesis, disrupting mitochondrial functions, and by producing oxidative stress (due to generation of free radicals). The primary tissues affected by VRA are intestinal and gastric mucosa, bone marrow, and spleen. VRA is a small, amphipathic molecule that can move passively across cell membranes. Trichothecenes are frequently found in buildings with water damage but can also be found in contaminated grain. Verrucarin A (VRA) is a macrocyclic trichothecene mycotoxin produced from Stachybotrys, Fusarium, and Myrothecium. Studies have also shown that OTA is present in sweat, which supports the use of sauna as a treatment to increase the excretion of OTA. Bentonite or zeolite clay is reported to reduce the absorption of multiple mycotoxins found in food, including OTA.
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Antioxidants such as vitamins A, E, C, NAC, rosmarinic acid, and liposomal glutathione alone or in combination have been shown to mitigate the oxidative effects of the toxin. Agents such as oral cholestyramine, charcoal, and phenylalanine can help prevent the absorption of these toxins from food. Treatment should be aimed at removing the source of exposure. Some studies have hypothesized that OTA may contribute to the development of neurodegenerative diseases such as Alzheimer's and Parkinson's. Dopamine levels in the brain of mice have been shown to be decreased after exposure to OTA. Studies have shown that OTA can lead to significant oxidative damage to multiple brain regions and is highly nephrotoxic. OTA can lead to kidney disease and adverse neurological effects. Exposure to OTA can also come from inhalation exposure in water-damaged buildings. Exposure is primarily through contaminated foods such as cereals, grape juices, dairy, spices, wine, dried vine fruit, and coffee. This chemical is produced by molds in the Aspergillus and Penicillium families. Ochratoxin A (OTA) is a nephrotoxic, immunotoxic, and carcinogenic mycotoxin. Here's the text that accompanies each positive result (bold emphasis mine): I'm wondering if I should get an MRI with Neuroquant before beginning treatment. With complicated cases, she ends up referring patients to Sonia Rapaport, MD, who is Shoemaker trained (but no longer listed on ). She is not Shoemaker trained, but she has helped many people with mold illness. My next appointment with my FM practitioner is on December 19. But what if my home is mold contaminated? Scary (and expensive) stuff to consider. I’ll need to get our home tested with ERMI, but hopefully it’s fine. I hope it's all connected.īoth types of mycotoxins could be caused by exposure to a water-damaged building, but also other things.
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I also have an odd pattern of thyroid dysfunction. I have no doubt that I have some degree of mitochondrial dysfunction. These mycotoxins could potentially be responsible for all of my symptoms! That means my symptoms might finally be addressed! I have a hyperactive immune system with many food sensitivities (and I just became sensitive to macadamia nuts, much to my chagrin), chronic constipation, gastritis/GERD, early satiety, insomnia, mental fatigue, difficulty concentrating, and impaired memory (my memory is nothing like it used to be, but I'd probably do fine on basic testing). I'm feeling a mixture of relief and fear. Great Plains Mycotoxins test results 11-23-17.PDF